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Biomedicines, Free Full-Text
SARS-CoV-2 spike protein induces inflammation via TLR2-dependent activation of the NF-κB pathway
SARS-CoV-2 spike protein as a bacterial lipopolysaccharide delivery system in an overzealous inflammatory cascade
Viruses, Free Full-Text
Frontiers Multifaceted role of SARS-CoV-2 structural proteins in lung injury
Host cell entry mediators implicated in the cellular tropism of SARS‑CoV‑2, the pathophysiology of COVID‑19 and the identification of microRNAs that can modulate the expression of these mediators (Review)
SARS-CoV-2 spike and nucleocapsid proteins fail to activate human dendritic cells or γδ T cells
Innate immune recognition against SARS-CoV-2, Inflammation and Regeneration
SciELO - Brasil - In vitro immunogenic profile of recombinant SARS-CoV2 S1-RBD peptide in murine macrophage and microglial cells In vitro immunogenic profile of recombinant SARS-CoV2 S1-RBD peptide in murine macrophage and
SARS-CoV-2 Spike protein is not pro-inflammatory in human primary macrophages: endotoxin contamination and lack of protein glycosylation as possible confounders
SARS-CoV-2 spike protein induces a differential monocyte activation that may contribute to age bias in COVID-19 severity
Potential Alternative Receptors for SARS-CoV-2-Induced Kidney Damage: TLR-4, KIM-1/TIM-1, and CD147
Inhibition of multiple SARS-CoV-2 variants entry by Lycium barbarum L. polysaccharides through disruption of spike protein-ACE2 interaction - ScienceDirect
Exploring the immunogenic properties of SARS-CoV-2 structural proteins: PAMP:TLR signaling in the mediation of the neuroinflammatory and neurologic sequelae of COVID-19 - ScienceDirect
SARS-CoV-2 spike protein induces the cytokine release syndrome by stimulating T cells to produce more IL-2
