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A patient with Crohn’s disease says his urine has become dark coloured and wonders if he should be concerned. What might be happening? IgA nephropathy
Extraglomerular origin. A history of haematuria, enuresis, dysuria, abdominal pain & fever is indicative of UTI.
What might be happening. IgA nephropathy.
She is angry and thinks the antibiotic has caused him harm. What do you say .
What other examinations would you perform: Vitals, especially BP Detailed skin inspection (why ) Abdominal examination to look for palpable kidneys (Wilm’s tumour, hydronephrosis, cystic kidney diseases, carcinoma) DRE (BPH, prostate CA) Urine dipstick test.
The mother complains that the urine has become a little darker and infrequent but puts this down to dehydration. What may be happening here. IgA nephropathy with Henoch–Schönlein purpura.
He thinks loud music is the cause. Of course you first thought of performing a DRE but instead went for the second best option of taking a urine sample. The dipstick indicates haematuria. What might be happening. Alport syndrome.
His pockets are full of bloody tissues. Following catheterisation a small amount of dark urine is produced. Describe this condition: Goodpasture syndrome Rapidly progressive (crescentic) glomerulonephritis Type I anti-GBM antibodies.
It just started after he passed bloody urine. What might be happening. What are other causes of non-glomerular haematuria. Urolithiasis most likely from excessive calcium intake Trauma (including masturbation), strenuous exercise, foreign body (kids & weirdos), UTI, sickle cell disease, tumours, drugs.
Foods: beets, blackberries, rhubarb Drugs: senna, antipsychotics, rifampicin Illness: Porphyria, hepatitis
ASO – antistreptolysin O (post strep glomerulonephritis), MPGN: membranoproliferative glomerulonephritis
VCUG: voiding cystourethrogram UPJ: Uretopelvic junction PUV: posterior urethral valves MCKD: medullary cystic kidney disease VUR: vesicoureteral reflux
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